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Lipopolysaccharide-induced tumor necrosis factor-α factor enhances  inflammation and is associated with cancer (Review)
Lipopolysaccharide-induced tumor necrosis factor-α factor enhances inflammation and is associated with cancer (Review)

Frontiers | The Balance of TNF Mediated Pathways Regulates Inflammatory  Cell Death Signaling in Healthy and Diseased Tissues
Frontiers | The Balance of TNF Mediated Pathways Regulates Inflammatory Cell Death Signaling in Healthy and Diseased Tissues

β-Ionone attenuates LPS-induced pro-inflammatory mediators such as NO, PGE2  and TNF-α in BV2 microglial cells via suppression of the NF-κB and MAPK  pathway - ScienceDirect
β-Ionone attenuates LPS-induced pro-inflammatory mediators such as NO, PGE2 and TNF-α in BV2 microglial cells via suppression of the NF-κB and MAPK pathway - ScienceDirect

Frontiers | P7C3 Inhibits LPS-Induced Microglial Activation to Protect  Dopaminergic Neurons Against Inflammatory Factor-Induced Cell Death in  vitro and in vivo
Frontiers | P7C3 Inhibits LPS-Induced Microglial Activation to Protect Dopaminergic Neurons Against Inflammatory Factor-Induced Cell Death in vitro and in vivo

TNF-α Induction by LPS Is Regulated Posttranscriptionally via a  Tpl2/ERK-Dependent Pathway: Cell
TNF-α Induction by LPS Is Regulated Posttranscriptionally via a Tpl2/ERK-Dependent Pathway: Cell

Methionine Attenuates Lipopolysaccharide-Induced Inflammatory Responses via  DNA Methylation in Macrophages | ACS Omega
Methionine Attenuates Lipopolysaccharide-Induced Inflammatory Responses via DNA Methylation in Macrophages | ACS Omega

Innate Immunity in Alcohol Liver Disease | IntechOpen
Innate Immunity in Alcohol Liver Disease | IntechOpen

Activation of Nur77 in microglia attenuates proinflammatory mediators  production and protects dopaminergic neurons from inflammation‐induced cell  death - Liu - 2017 - Journal of Neurochemistry - Wiley Online Library
Activation of Nur77 in microglia attenuates proinflammatory mediators production and protects dopaminergic neurons from inflammation‐induced cell death - Liu - 2017 - Journal of Neurochemistry - Wiley Online Library

Genkwanin Inhibits Proinflammatory Mediators Mainly through the Regulation  of miR-101/MKP-1/MAPK Pathway in LPS-Activated Macrophages | PLOS ONE
Genkwanin Inhibits Proinflammatory Mediators Mainly through the Regulation of miR-101/MKP-1/MAPK Pathway in LPS-Activated Macrophages | PLOS ONE

Fucoxanthin attenuates LPS-induced acute lung injury via inhibition of the  TLR4/MyD88 signaling axis | Aging
Fucoxanthin attenuates LPS-induced acute lung injury via inhibition of the TLR4/MyD88 signaling axis | Aging

LPS-Induced Rodent Sepsis Model - Creative Biolabs
LPS-Induced Rodent Sepsis Model - Creative Biolabs

Protopine attenuates inflammation stimulated by carrageenan and LPS via the  MAPK/NF-κB pathway - ScienceDirect
Protopine attenuates inflammation stimulated by carrageenan and LPS via the MAPK/NF-κB pathway - ScienceDirect

Diagram of a putative LPS-induced PTP/LITAF/TNF signaling pathway. |  Download Scientific Diagram
Diagram of a putative LPS-induced PTP/LITAF/TNF signaling pathway. | Download Scientific Diagram

Inhibition of Lipopolysaccharide-Induced Inflammatory and Oxidative  Responses by Trans-cinnamaldehyde in C2C12 Myoblasts
Inhibition of Lipopolysaccharide-Induced Inflammatory and Oxidative Responses by Trans-cinnamaldehyde in C2C12 Myoblasts

TNF-alpha阻害 | TNF-alpha Inhibition
TNF-alpha阻害 | TNF-alpha Inhibition

A Novel Carboline Derivative Inhibits Nitric Oxide Formation in Macrophages  Independent of Effects on Tumor Necrosis Factor α and Interleukin-1β  Expression | Journal of Pharmacology and Experimental Therapeutics
A Novel Carboline Derivative Inhibits Nitric Oxide Formation in Macrophages Independent of Effects on Tumor Necrosis Factor α and Interleukin-1β Expression | Journal of Pharmacology and Experimental Therapeutics

GFI-1 Protects Against Lipopolysaccharide-Induced Inflammatory Responses  and Apoptosis by Inhibition of the NF-κB/TNF-α Pathway in H9c2 Cells |  SpringerLink
GFI-1 Protects Against Lipopolysaccharide-Induced Inflammatory Responses and Apoptosis by Inhibition of the NF-κB/TNF-α Pathway in H9c2 Cells | SpringerLink

Molecules | Free Full-Text | Chlojaponilactone B Attenuates  Lipopolysaccharide-Induced Inflammatory Responses by Suppressing  TLR4-Mediated ROS Generation and NF-κB Signaling Pathway
Molecules | Free Full-Text | Chlojaponilactone B Attenuates Lipopolysaccharide-Induced Inflammatory Responses by Suppressing TLR4-Mediated ROS Generation and NF-κB Signaling Pathway

Figure 1 | A Retrospective on Nuclear Receptor Regulation of Inflammation:  Lessons from GR and PPARs
Figure 1 | A Retrospective on Nuclear Receptor Regulation of Inflammation: Lessons from GR and PPARs

Signaling pathways involved in LPS induced TNFalpha production in human  adipocytes | Journal of Inflammation | Full Text
Signaling pathways involved in LPS induced TNFalpha production in human adipocytes | Journal of Inflammation | Full Text

A Novel Strategy for TNF-Alpha Production by 2-APB Induced Downregulated  SOCE and Upregulated HSP70 in O. tsutsugamushi-Infected Human Macrophages |  PLOS ONE
A Novel Strategy for TNF-Alpha Production by 2-APB Induced Downregulated SOCE and Upregulated HSP70 in O. tsutsugamushi-Infected Human Macrophages | PLOS ONE

Lipopolysaccharide-induced inflammatory liver injury in mice - K Hamesch, E  Borkham-Kamphorst, P Strnad, R Weiskirchen, 2015
Lipopolysaccharide-induced inflammatory liver injury in mice - K Hamesch, E Borkham-Kamphorst, P Strnad, R Weiskirchen, 2015